Wednesday, February 20, 2019
Critical Review of the Literature of the role that Dietary Factors Play in Preventing Type 2 Diabetes Essay
Critical round off of the Literature of the role that viandsetic Factors Play in Preventing geek 2 DiabetesIntroductionType 2 Diabetes (T2D) is a rapidly growing international public wellness issue. It has been report that 285 million 2079 year olds had the sickness in 2010 global and this is estimated to grow to 439 million by 2030. (19) T2D has been associated with a variety of another(prenominal) health problems such(prenominal) as cardiovascular disease, blindness and shortened life expectancy. (4,40) The prevalence of the disease is associated with obesity and over cargo as well as a westbound viandsetic pattern and lack of exercise. (17) It has been reported that approximately 80% of people who develop T2D atomic number 18 obese or over weight former to diagnosis.(8) Diabetes is a go onable disease, (19) with weight loss beingness identified as one of the approximately powerful interventions. (4) Diet and natural activity (PA) atomic number 18 potent interve ntions in attaining this (25) and gravel withal been linked with decline in run a hazard independently.(40) The primary focus of this newspaper impart look at provender and recap the shew on whether food alone sewer second to prevent T2D. As a secondary theme it ordain in like manner look at the evidence that forage impart function to prevent the develop custodyt of and help to control symptoms aft(prenominal) T2D has been diagnosed. 8 paper will reviewed, (26-33) with a purpose of identifying some practical, evidence based forageetical guidelines. dietetic guidelines argon easier to fol pitiable when they sign unique(p cherryicate) foods and patterns rather than nutrients and properties of foods and greater compliance can be achieved . (29,31) For this reason the studies reviewed focus on particular(prenominal) foods and diet geniuss rather than macronutrients and or micronutrients which do non translate into dietary guidelines as easily.Literature Sear chA literature review utilize the interest selective informationbases was carried turn tabu Pubmed, Web of Science, Embase, CINAHL, British Nursing Index and Medline. The fol beginninging keywords were employ in various combinationsDiet, prevention, food, T2D, interventions, dietary, harvest-tide, veggies, nutrition. Other resources such as Google pupil and NHS Choices behind the headlines were excessively utilised. Be form T2D is an international issue, papers from around the world were considered. The criteria for inclusion body was slight than 10 years old, the intervention had to be a specific food collection or diet style, measuring new incidents of T2D, or changes is symptoms associated with T2D and adults as the sample throng.Diet as a official document to prevent T2D6 Studies looked at whether diet can prevent T2D. Please worry to Table 1 for details of all the studies. Villegas et al (26) and Bazzano et al (27) looked at take, veg and fruit juices wasting disease and fruit and vegetable consumption respectively. some(prenominal) of the studies utilise women only in their sample stems, limiting the generalizability of the findings to the wider population. (5) dietetical sagaciousness was achieved finished Food Frequency Questionnaires (FFQ) in some(prenominal) studies. Villegas et al (26) stick outd evidence of their FFQ having been validated, (21) however reported using it only 3 clock at bottom a 4.6 year timeframe, questioning the trueness of their self-collected selective information. Bazzano et al (27) data collection was retrospective in that the FFQ was designed in 1984 and fol emited up at 4 year intervals. The concern with such a dated questionnaire is that it has not accounted for different trends that hand over occurred over time, affecting the accuracy of the results.Villegas et al (26) substantiate incidence of T2D through subjects meeting the criteria set out by the American Diabetic acquaintance.(2) Bazzan o et al (27) used criteria set out by guinea pig Diabetes Data meeting for all participants up to and including 1997.(16) The Criteria for participants after 1998 was set out by the American Diabetic Association. The reason for this was due to new criteria being print at this time. (2) The main difference being the plasma glucose variant changing from 7.8mmol/l or much to 7.0mmol/l or much. (9) The Criteria peck out by the World wellness Organisation in 1985, (24) could deplete been used to confirm incidence of T2D in the Bazzano et al (27) mull . It was published 1 year after baseline data was taken and would prep are resulted in a steep percentage of participants being diagnosed with the similar criteria, annex consistencyand reliability in the results.Villegas et al (26) reported that a proud consumption of vegetables was associated with a subordinated take a chance of T2D. Participants who had a higher(prenominal) vegetable breathing in were in any case less apt(predicate) to shutout and have higher directs of PA, both factors that can reduce the bump of T2D questioning the causality of the reduction in risk. (4) Participants with a higher fruit intake were also less likely to quite a little and have higher levels of PA. There was no knowledge made in the midst of fruit intake and risk of T2D thitherfore it is practical to consider that con rearers like PA and smoking whitethorn not have resultant roleed the level of risk to T2D in this battlefield.Bazzano et al (27) reported an opponent connectedness between whole fruit and green silky-leaved vegetable intake. Women who had a higher intake of fruit and vegetables were older, less likely to smoke and more(prenominal) than likely to have higher levels of PA. harvest Juice was positively associated with incidence of T2D which could be due to high sugar content.. (22) Also participants who had the highest fruit juice intake had the lowest levels of PA which is associated with change magnitude risk of T2D. (20)Salas-Salvado et al (28) and Martinez-Gonzalez et al (29) both studied the risk of T2D and adherence to the Mediterranean diet (MedDiet). A MedDiet is characterised by high consumption of fruit, vegetables, whole grains, olive oil color, nuts, pulses, tip and reduced consumption of red and elegant middles, high fat dairy refined gains and foods high in sugar and starch. (11) Salas-Salvado et al (28) compared adherence to a MedDiet supplemented with either olive oil or nuts with a low fat diet (control group).The supplemented items in the MedDiets were given to participants. Participants in the control group were given non dietary gifts to encourage adherence. There are concerns about the ethics of using incentives and gifts in research. (6)Within this reputation the use of gifts appears innocuous, the concern lies with how adherence to the diet is reliably measured. When participants are given vital ingredients, this will influence their dietary intake, making it difficult generalize the results. (13) The sample population in this study were older and had at least 3 risk factors relating to cardiovascular disease again making it more difficult to generalise results. Participants were given 7 goals, includingincreasing vegetable and fruit consumption, minify red and elegant meat and increasing the consumption of either olive oils or nuts.The control group were asked to reduce all types of fat. Results showed that participants in the MedDiet groups had a greater reduced risk of T2D. chance was reduced by 51% in the olive oil group and 52% in the nut group. These findings are backed up by other studies. (38-39) Diabetes incidence was lour in those who attained 4 of the 7 goals. PA levels and changes in weight did not differ through all 3 groups, although the participants in the both the MedDiet groups were associated with higher levels of PA. This study was carried out on Spanish participants, who traditionally f ollow a MedDiet. The control group may have had a strong adherence to a MedDiet naturally, which could advert on the reliability of the results.Martinez-Gonzalez et al (29) used participants who were nurses and university graduates. preponderance in T2D is associated with lower socioeconomic status, (1) so by using the participants from a higher socioeconomic group may bias the findings. (13) Participants were not excluded if they had Diabetes at baseline. Data was collected via FFQ which consisted of 136 items with 9 responses ranging from never to more than 6 times a day. Points were allocated to crack a score indicating level of adherence. Questions covered areas such as formulation methods, supplements and fats and oils. This FFQ goes into a lot of detail to obtain the most information it can about participants diets, increasing the validity of the data.However reliability is compromised as participants are asked to recall food intake from the previous year decreasing the ac curacy of data collected. (5) The results indicated a significant reduction in risk of ontogeny T2D in those who with strong adherence to the MedDiet after adjustment for age and sex. Participants with a score of 6 had an 83% reduction. Although participants in this group had the highest levels of physical activity, which is a known factor in decreasing the risk of T2D, they also had a higher baseline prevalence for increased risk factors for Diabetes such as age higher BMI and higher blood pressure. This adds weight to the findings the MedDiet can reduce the risk of developing T2D.Fung et al (30) and VanDam et al (31) looked at more talk dietarypatterns. VanDam et al (31) used a 131 item FFQ specifying specific foods, ingredient sizes and frequency of intake. oer a space of 12 years data on food intake was collected 3 times. In put together to make these results more legitimate data collection should have occurred more frequently. The participants were all male health profes sionals making the sample group sort of specific reducing the ability to generalise the findings to the wider population. (13) Foods were classified into groups based on nutritional profiles. Factor analysis was then applied in regularize to identify food patterns. Two dietary patterns were identified Prudent and westerly. Prudent was characterised by high consumption of vegetables, fish and whole grains and westerly by high consumption of red and processed meat, high-fat dairy and eggs. workforce with higher Western patterns were younger, more likely to smoke and did less PA. workforce with higher Prudent diet patterns were older less likely to smoke and engaged in more PA. The Prudent diet was associated with a lower-ranking reduction in risk in developing T2D with wholegrain foods having the highest inverse association. harvest-time and vegetables were not considerably associated with reduced risk. The Western diet was associated with considerably higher risk of developing T2D. Processed meat, other processed foods and refined grains indicating the most significant association. This could indicate that cutting out specific foods could me more skilful in reducing the risk of T2D than increasing intake of other foods.Fung et al (30) used participants from the Nurses Health Study which was established in 1976. This is the same study from which Bazzano et al (27) took their participants. The same FFQ was used in this study with baseline also being the 1984 FFQ as this was the expanded 116 item version. The information obtained was then used and classified in the same way as the VanDam et al (31) study producing the same Prudent and Western dietary patterns. The results from this study focus in the first place on the Western diet pattern. Similarly to the men in the VanDam et al (31) study, women who scored high in the Western diet pattern were more likely to smoke. The results also mirrored that of the VanDam et al (31) study in that it reported an inc reased risk of developing T2D and a Western diet pattern. This study investigated the characteristics of the Western dietary pattern further and found positive associations between red and processed meats andthe development of T2D.This could also add weight to the previous definition that cutting out specific foods, such as red and processed meats could be more beneficial than adding other food groups in preventing T2D. A replica study using the same FFQ and Prudent and Western diet pattern and using a sample group that consisted of both men and women could add strength to the finds of both of these studies.(5)Diet as a tool in preventing the development of and expectant greater control over the symptoms of T2DElhayany et al (32) compared a low carbohydrate Mediterranean diet (lowest common multiple) a traditional Mediterranean diet (TM) and the 2003 American Diabetic Association diet (ADA) on health parameters. Glycemic control for people with T2D diagnosis was one of the outcome measures. Participants were at random assigned to 1 of the 3 groups, given recommendations for daily intake on nutritional elements such as calories and protein and advised to engage in 30-45 proceeding of PA a week. The LCM and TM diets included only low glycemic index carbohydrates, with LCM having a lower %. The TM and ADA diets had the same % of carbohydrates but the ADA also included mixed glycemic index carbohydrates.FFQ were used asking the participants to recall the last 24hour food intake. It is felt this data will be more accurate than those studies asking participants to recall food intake from the previous year, making results more reliable. Data was collected 3 times over a 12 month period. Results showed all groups had reduced weight and BMI with no significant difference. All 3 dietary interventions reduced factors that increase glycemic control such as HbA1c and triglyceride levels. The LCM diet was the most effective in increasing glycemic control.Esposito et al ( 33) compared a LCM and a low fat calorie restricted diet (LFD) on glycemic control and the detainment on needing to commence antihyperglycemic practice of medicine in people newly diagnosed with T2D. The LCM diet was rich in fruit vegetables and whole grains and low in red meat. There was also a requirement that no more than 50% of calories was fromcarbohydrates. Complex carbohydrates rather than low GI carbohydrates were stipulated. Some interlacing carbohydrates can have high GI levels, which are associated with increased risk of Diabetes. (10) The LFD was based on American Heart Association guidelines. (12) Participants were randomized into 1 of 2 groups asked to keep food diaries and given guidelines on increasing physical activity.Data was collected through reviews of the diary. Food diaries could provide more reliable information than FFQ if they are filled out daily. There is still a risk that they could be filled out inaccurately, and that participants may modifies their intake as a result of keeping a diary. The study reported that both groups lost weight, but reduction was greater in the LCM. Overall thither was a significant difference in the need to commence antihyperglycemic medication between the LCM and the LFD 44% and 70% respectively. Potentially this result could have shown more significance if low GI carbohydrates were stipulated instead of complex carbohydrates. discussionThe findings coming out of this review indicate that certain dietary interventions perchance helpful in reducing the risk of developing T2D and may also help with glycemic control after diagnosis.(26-33) Some food groups and dietary patterns provide more tenacious evidence than others. The results regarding fruit intake and risk of T2D are inconsistent within this review with Villegas et al (26) reporting no association between fruit and risk reduction, where as Bazzano et al (27) did. extravagantly consumption of fruit and vegetables are a major component in the Med iterranean diet and the Prudent dietary pattern, both of which were associated with an inverse association. vegetables are more consistently associated with a reduced risk, in this review and a new-fashioned meta-analysis, (3) especially green leafy vegetables. (27) More research studying the set up of fruit and vegetables separately is needed. This inconsistency is reflected in other studies and systematic reviews. (7) High intake of fruit and vegetables has often been associated with higher PA levels within this review, (26-29,31) which is a risk reducing factor in itself.Part of the problem could be that many studies that look at dietary interventions are prospective age bracket studies andalthough they can provide an association they can not prove whether the ingest is due to the dietary item or another factor such as PA or weight loss. More experimental designed research is needed so that a direct cause between diet and the reduction of T2D can be established. (13) While it is unclear the minute role that fruit and vegetables play in reducing the risk of T2D there is an abundance of evidence that a diet rich in fruit and vegetables is beneficial in overall health (34-35) and weight reduction, (23) so including them as part of a healthy diet may indirectly help to reduce the risk of T2D.Red and processed meat has been more consistent in its positive association to developing T2D. Fung et al (28) and VanDam et al (29) found strong associations between consumption and increased risk of T2D. One of the characteristics of The Mediterranean diet is the absence or reduction of red and processed meats, and this diet has been associated with reduced risk. These findings were backed up in a recent meta-analysis paper (18) studying 3 cohorts whos conclusion educes that red meat consumption, particularly processed red meat is linked to higher risk of developing T2D.Meta-analysis produces level 1 evidence, providing increased confidence in the conclusions and goo d substructure for providing evidence based information such as dietary guidelines.(13) Diets high in red and processed meats are linked to high cholesterol (14) which is one of the leading causes of death in people with T2D (15) and other stark health conditions such as cancer and cardiovascular disease. (36) So date increasing fruit and vegetable intake may have a more beneficial and holistic effect on health, the same could be said for reducing the intake of red and processed.Salas-Salvado et al (28) produced higher level evidence being a randomised control trial giving more weight to their findings(5) succeeding(a) a MedDiet can reduce the risk of T2D. One of the studies that looked at the effect of diet after diagnosis, (32) was also a randomised control trial providing the same level of evidence and weight to their findings that LCM can delay the need for hyperglycaemic medication therefore having a positive beneficial effect on T2D. Although these two studies looked at t he effect of diet after diagnosis it could be reasonable to suggest that following the dietarypatterns associated with these findings, prior to diagnosis could help prevent T2D as they are similar dietary patterns to two of the other studies.(28-29)While the findings from this review indicate that making changes to ones diet may help to reduce the risk of Diabetes occurring, it also suggests that implementing the same kind of dietary changes may help with glucose control after Diabetes has been diagnosed, preventing the further development of the disease and the need for antihyperglycemic medication. (32-33) One study has implied that dietary changes in the form of energy travail can actually deform beta cell failure and insulin resistance, symptoms found in T2D.(37) It is a precise small study and the dietary intervention is severe energy restriction making it difficult to generalise to the wider population, but it is a controlled study and could provide interesting findings tha t further research could be built upon, that dietary interventions may be able to reverse the symptoms of T2D. Collating this information together it seems reasonable to suggest that making positive changes to dietary habits could have benefits pre and post T2D diagnosis.dietary GuidelinesThe findings from the 8 studies in this review can not all be discussed in detail due to word limitation. However recurring themes seem to be occurring, providing information on which to base a set of guidelines. Going by these findings the following guidelines are recommendedReduce intake of red meat and processed meat (all colours)Substitute these with white meats and fishReduce intake of other processed foodsReduce intake of refined grainsReduce intake of high sugar foods and drinks including fruit juices Carbohydrate intake should be based on items with a low GI score extend intake of olive oilIncrease nut intakeIncrease intake of pulsesIncrease wholegrain intakeIncrease vegetable intake espec ially green leafy vegetablesIncrease fruit intakeA Mediterranean style diet is characterised by much of this advise and is therefore a recommended diet style to follow.ConclusionThe prevalence of T2D is growing around the world. It has been associated with many other health problems and reduces quality of life and life expectancy. It is a preventable disease and diet is one of the ways in which this disease can be combated. Dietary guidelines have been recommended from the findings of this review, based on following a Mediterranean diet, reducing intake of red and processed meats and other processed foods and increasing intake of foods such as fruit and vegetables, wholegrains and olive oils and nuts. While it has been acknowledged that more research needs to be carried out to further examine the cause and effect between diet and T2D, it is reasonable to suggest that one may find these dietary changes beneficial in helping to reduce the risk of T2D and other areas of health, possibl y helping to indirectly reduce risk of T2D. It is also reasonable to suggest that a change in diet may bring beneficial changes once diagnosis has been given.Table 1 annex list1. Agardh E, Allenbeck P, Hallqvist J, Moadi T and Sidorchuk A. Type 2 Diabetes and Socioeconomic Position A Systematic Review and Meta-analysis. International Journal of Epidemiology. 2011 40(3) 804-8182. American Diabetic Association subject field of the Expert Committee on the Diagnosis and Classification of Diabetes Mellitus. Diabetes Care. 2000 23(1s)3. Carter P, color LJ, Troughton J, Khunti K, and Davies M. Fruit and vegetable intake and incidence of Type 2 Diabetes mellitus A Systematic Review and Meta-analysis. British health check Journal. 2010 341c42294. Davis MJ, Tringham JR, Troughton J, Kunit KK. Prevention of T2D mellitus. A review of the Evidence and its Application in a UK Setting. Diabetic Medicine. 2004 21 403-4145. Gerrish K and Lacey A. The Research Process in Nursing. 2006 5th Edition. Oxford Blackwell Publishing.6. Grant RW and Sugarman J. Ethics in Human Subjects Research Do Incentives Matter? Journal of Medicine and Philosophy. 2004 29(6) 717-7387. Hamer M, Chida Y. wasting disease of Fruit and Vegetables and Antioxidants and Risk of Type 2 Diabetes. A Systematic Review and Meta-analysis. Journal of Hypertension. 2007 252361-23698. Hensrud DD. Dietary Treatment and Long-term Weight wrong and Maintenance in T2D. Obesity Research. 20019(4 supplement)348S-353S9. Hickner RC, Brunson MA, McCammon M, Mahar MT, Garry JP, Houmard JA. Diabetic Groups as Defined by ADA and NDDG Criteria have a Similar Aerobic Capacity, Blood obligate and Body Composition. American Diabetes Association and issue Diabetes Data Group. Diabetologica. 2001 Jan 44(1) 26-3210. Hodge AM, ODea K, side DR, and Giles GG. Glycemic Index and Dietary Fibre and the Risk of Type 2 Diabetes. Diabetes Care. 2004 27(11) 2701-270611. Kastorini MC, Milionis HJ, Esposito K, Giugliano D, Goudevenos JA, pa nagiotakos. The proceeds of Mediterranean Diet on Metabolic Syndrome and its Components. Journal of the American College of Cardiology 2011 57(11) 1299-131312. Krauss RM, Eckle RH, Howard B, Appel LJ, Daniels SR, and Deckelbaum RJ.AHA Dietary Guidelines Revision 2000 A Statement for Healthcare Professionals from the Nutrition Committee of the American Heart Association. Circulation. 2002 1022284-9913. LoBiondo-Wood G. and Haber J. Nursing Research. Methods and Critical Appraisal for Evidence Based Practice. 2006 sixth Edition. Mosby Missouri14. Micha R, Wallace SK, and Mozaffarian MD. Epidemiology and Prevention. Red and Processed shopping centre employment and Risk of consequent Coronary Heart Disease, Stroke and Diabetes Mellitus. A systematic Review and Meta-Analysis. American Heart Association. 2010 121 2271-228315. Heart Disease and Stroke The Nations leading Killers. At a Glance National Centre for Chronic Disease Prevention and Health Promotion. Division for Heart Disease and Stroke prevention. 2011 CS217229-AI16. National Diabetes Data Group Classifications and Diagnosis of Diabetes Mellitus and Other Categories of Glucose Intolerance. Diabetes. 1979 28 1039-105717. Neild L, Summerbell CD, Hooper L, Whittaker V, Moore H. Dietary Advice for the Prevention of T2D Mellitus in Adults (Review) The Cochrane Collaboration. 2008 Wiley18. Pan A, Sun Q, Bernstein AM, Schulze MB, Manson JE, Willett WC and Hu FB. Red Meat Consumption and Risk of Type 2 Diabetes 3 Cohorts of US Adults and an Updated Meta-analysis. American Journal of Clinical Nutrition. 2011 94(4) 1088-109619. Shaw JE, Sicree RA, Zimmet PZ. Global Estimates of the Prevalence of Diabetes for 2010 and 2030. Diabetes Research in Clinical Practice. 2010874-1420. Sigal RJ, Kenny GP, Wasserman DH, Castaneda-Sceppa C and White RD. physiologic Activity/Exercise and Type 2 Diabetes. A Consensus Statement fromthe American Diabetes Association. Diabetes Care. 2006 29(6) 1433-143821. Shu XO, Yang G, Jin F , Liu D, Kushi L, Wen W, Gao YT, Zheng W. Validity and Reproducibility of the Food Frequency Questionnaire Used in the Shanghai Womens Health Study. European Journal of Clinical Nutrition. 200458172322. Shulze MB, Manson JE, Ludwig DS, Colditz GA, Stampfer MD, Willet WC, and Hu FB. Sugar-Sweetened Beverages, Weight Gain, and Incidence of Type 2 Diabetes in Young and Middle-Aged Women. The Journal of the American Medical Association. 2004 292(8)927-93423. Tohill B, Seymour J, Serdula M, Kettle-Khan L, and Rolls BJ. What epidemiologic Studies Tell Us about the Relationship between Fruit and Vegetable Consumption and Body Weight. Nutrition Reviews. 2004 365 -37424. WHO Study Group on Diabetes Mellitus. Diabetes Mellitus Report of a WHO Study Group. Geneva World Health Organisation 198525. overnice Public Health Guidance. Preventing Type 2 Diabetes Population and Community Interventions. National Institute for Health and Clinical Excellence. 2011 Issue 3526. Villagers R, Shu OX, Gao YT , Yang G, Elasy T, Li H and Zheng W. Vegetable but Not Fruit Consumption Reduces the Risk of Type 2 Diabetes in Chinese Women. The Journal of Nutrition. 2008 138 574-58027. Bazzano LA, Kamudi JJ, Hu FB, and Li TY. Intake of Fruit, Vegetables and Fruit Juices and Risk of Diabetes in Women. Diabetes Care. 2008 31(7) 1311-131728. Salas-Salvado J, Bullo M, Babio N, Martinez-Gonzalez MA, Jurado NI, Basora J, Estruch R, Covas MI, Corella D, Aros F, Gutierrez VR, and Ros E. Reduction in the Incidence of Type 2 Diabetes With the Mediterranean Diet. Diabetes Care. 2011. 3414-1929. Martinez-Gonzalez MA, Fuente-Arrillaga CDL, Nunez-Cordoba JM, Basterra-Gotari FJ, Beunza JJ, Vazquez Z, Benito S, Tortosa A and Bes-Rasrollo M. Adherence to Mediterranean Diet and Risk of Developing Diabetes Prospective Cohort Study. British Medical Journal. 2008 336135130. Fung TT, Schulze M, Manson JE, Willet WC, and Hu FB. Dietary Patterns, Meat Intake and the Risk of Type 2 Diabetes in Women. annals of Interna tional Medicine. 20041642235-224031. VanDam RM, Rimm EB, Willet WC, Stampfer MJ and Hu FB. Dietary Patterns and Risk for Type 2 Diabetes Mellitus in US Men. Annals of Internal Medicine. 2002136 201-20932. Elhayany A, Lustman A, Abel R, Attal-Singer J and Vinker S. A Low Carborhydrate Mediterranean Diet Improves Cardiovascular Risk Factors and Diabetes Control Among Overweight Patients with Type 2 Diabetes Mellitus A 1-year Prospective randomized intervention Study. Diabetes, Obesity and Metabolism. 2010 12204-20933. Esposito K, Maiorino IM, Ciotola M, Palo CD, Scognamiglio P, Gicchino M, Petrizzo M, Saccomanno F, Beneduce F, Ceriello A and Guigliano D. Effects of a Mediterranean-Style Diet on the Need for Antihyperglycemic Drug Therapy in Patients with Newly Diagnosed Type 2 Diabetes. Annals of Internal Medicine. 2009 151 306-31434. Van Duyn MAS and Pivonka E. Over view of the health benefits of fruit and vegetable consumption for the Dietetics Professional. Journal of the American Dietetic Association. 2000 100(12) 1511-152135. Anderdson JW, Baird P, Davis RH, Ferreri S, Knudtson M,Koraym A, Waters V,and Williams CL. Health Benefits of Dietary Fibre. Nutrition Reviews. 2009 67(4) 188-20536. Sinha R, Cross AJ, Graubaed BI, Leitzmann MF, and Schatzin A. Meat Intake and Mortality. A Prospective Study of Over Half a Million People.Archives of Internal Medicine. 2009 169(6) 562-57137. Lim EL, Hollingsworth KG, Aribisala BS, Chen MJ, Mathers JC and Taylor R. Reversal of type 2 Diabetes Normalisation of Beta Cell Function in Association with diminish Pancreas and Liver Triacylglycerol. Diabetologica. 2011 542506-251438. Jiang R, Manson JE, Stampfer MJ, Liu S, Willet WC, Hu FB. Nut and Peanut Butter Consumption and Risk of Type 2 Diabetes in Women. Journal of the American Medical Association. 2002 28(20) 2554-256039. Riserus U, Willet WC, and Hu FB. Dietary Fats and Prevention of Type 2 Diabetes. Progress in Lipid Research. 2009 48(1)44-5140. Tuomilehto J, Lindstrom J, Eriksson JG, Valle TT, Hamalainen H, Ilanne-parikka P, Keinanen-Kiukaanniemi S, Laakso M, Louheranta A, Rastas M, Salminen V and Uusitupa M. Prevention of Type 2 Diabetes Mellitus by Changes in Lifestyle among Subjects with Impaired Glucose Intolerance. The New England Journal of Medicine. 2001 344(18) 1343-1350
Subscribe to:
Post Comments (Atom)
No comments:
Post a Comment